Pulmonary arteriovenous malformations may be the only clinical criterion present in genetically confirmed hereditary haemorrhagic telangiectasia

Emily Anderson, Lakshya Sharma, Ali Alsafi, Claire L Shovlin

Research output: Contribution to journalArticlepeer-review

Abstract

Pulmonary arteriovenous malformations (PAVMs) result in preventable complications demanding specialty care. Underlying hereditary haemorrhagic telangiectasia (HHT) can be identified by genetic testing, if the diagnosis is considered. Retrospectively reviewing 152 unrelated adults with genetically confirmed HHT due to ACVRL1, ENG or SMAD4, we found that only 104/152 (68%) met a clinical diagnosis of HHT with three Curaçao criteria. The genetic diagnostic rate was similar for patients with three (104/137, 76%) or one to two (48/71, 68%; p=0.25) criteria. Of 83 unrelated probands with PAVM(s) and genetically-confirmed HHT, 20/83 (24%) had few, if any, features of HHT. Enhanced clinical suspicion, as well as HHT genetic testing, is recommended if one or more PAVMs are present.

Original languageEnglish
Pages (from-to)628-630
Number of pages3
JournalThorax
Volume77
Issue number6
DOIs
Publication statusPublished - 14 Feb 2022

Keywords

  • Activin Receptors, Type II/genetics
  • Adult
  • Arteriovenous Fistula
  • Arteriovenous Malformations/complications
  • Humans
  • Pulmonary Artery/abnormalities
  • Pulmonary Veins/abnormalities
  • Retrospective Studies
  • Telangiectasia, Hereditary Hemorrhagic/diagnosis

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