Abstract
Recent studies have identified at least nine distinct receptors used by the picornaviruses for cell entry. Does the evolution of receptor usage correlate with the different tropisms observed in this group of viruses, and does this influence pathogenesis, or is it the consequence of another selection mechanism that favours virus survival?.
| Original language | English |
|---|---|
| Pages (from-to) | 198-202 |
| Number of pages | 5 |
| Journal | Trends in Microbiology |
| Volume | 6 |
| Issue number | 5 |
| Publication status | Published - May 1998 |
UN SDGs
This output contributes to the following UN Sustainable Development Goals (SDGs)
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SDG 3 Good Health and Well-being
Keywords
- DECAY-ACCELERATING FACTOR
- HUMAN POLIOVIRUS RECEPTOR
- FACTOR CD55
- ENCEPHALOMYOCARDITIS VIRUS
- HUMAN RHINOVIRUS
- TRANSGENIC MICE
- VITRONECTIN RECEPTOR
- COXSACKIEVIRUS A9
- NASAL SECRETIONS
- HEPARAN-SULFATE
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